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Julia R. Koehler, MD
Senior Associate Physician in Pediatrics, Division of Infectious Diseases
Associate Professor of Pediatrics, Harvard Medical School
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Julia R. Koehler, MD
Senior Associate Physician in Pediatrics, Division of Infectious Diseases
Associate Professor of Pediatrics, Harvard Medical School
Medical Services
Languages
English
Education
Medical School
University of Heidelberg
1985
Heidelberg
Germany
Internship
Children’s Hospital of University of Heidelberg
Heidelberg
Germany
Residency
Pediatrics
Boston Children's Hospital
1993
MA
Fellowship
Pediatric Infectious Diseases
Boston Children's Hospital
1998
Boston
MA
Certifications
American Board of Pediatrics (General)
American Board of Pediatrics (Infectious Diseases)
Publications
Candida albicans' inorganic phosphate transport and evolutionary adaptation to phosphate scarcity. View Abstract
Glycerophosphocholine provision rescues Candida albicans growth and signaling phenotypes associated with phosphate limitation. View Abstract
Effect of clinician information sessions on diagnostic testing for Chagas disease. View Abstract
Stress- and metabolic responses of Candida albicans require Tor1 kinase N-terminal HEAT repeats. View Abstract
Inflammasome-mediated GSDMD activation facilitates escape of Candida albicans from macrophages. View Abstract
Perceived barriers to Chagas disease screening among a diverse group of prenatal care providers. View Abstract
Trends in Pediatric Candidemia: Epidemiology, Anti-Fungal Susceptibility, and Patient Characteristics in a Children's Hospital. View Abstract
Phosphate in Virulence of Candida albicans and Candida glabrata. View Abstract
Phosphoric Metabolites Link Phosphate Import and Polysaccharide Biosynthesis for Candida albicans Cell Wall Maintenance. View Abstract
Medical Deferred Action - Living on Borrowed Time. View Abstract
Global Transcriptomic Analysis of the Candida albicans Response to Treatment with a Novel Inhibitor of Filamentation. View Abstract
Candida albicans Dispersed Cells Are Developmentally Distinct from Biofilm and Planktonic Cells. View Abstract
Intersection of phosphate transport, oxidative stress and TOR signalling in Candida albicans virulence. View Abstract
Hepatic Legionella pneumophila Infection in an Infant With Severe Combined Immunodeficiency. View Abstract
The Human Gut Microbial Metabolome Modulates Fungal Growth via the TOR Signaling Pathway. View Abstract
The Candida albicans TOR-Activating GTPases Gtr1 and Rhb1 Coregulate Starvation Responses and Biofilm Formation. View Abstract
Skin infections are eliminated by cooperation of the fibrinolytic and innate immune systems. View Abstract
Fungi that Infect Humans. View Abstract
Phosphate is the third nutrient monitored by TOR in Candida albicans and provides a target for fungal-specific indirect TOR inhibition. View Abstract
Beauvericin Potentiates Azole Activity via Inhibition of Multidrug Efflux, Blocks Candida albicans Morphogenesis, and Is Effluxed via Yor1 and Circuitry Controlled by Zcf29. View Abstract
Antagonism of Fluconazole and a Proton Pump Inhibitor against Candida albicans. View Abstract
Ribosomal protein S6 phosphorylation is controlled by TOR and modulated by PKA in Candida albicans. View Abstract
The silent crisis: children hurt by current immigration enforcement policies. View Abstract
Physiologic expression of the Candida albicans pescadillo homolog is required for virulence in a murine model of hematogenously disseminated candidiasis. View Abstract
Dispersion as an important step in the Candida albicans biofilm developmental cycle. View Abstract
Harnessing Hsp90 function as a powerful, broadly effective therapeutic strategy for fungal infectious disease. View Abstract
The Candida albicans pescadillo homolog is required for normal hypha-to-yeast morphogenesis and yeast proliferation. View Abstract
Mucosal damage and neutropenia are required for Candida albicans dissemination. View Abstract
CaNAT1, a heterologous dominant selectable marker for transformation of Candida albicans and other pathogenic Candida species. View Abstract
Lung epithelial cells and extracellular matrix components induce expression of Pneumocystis carinii STE20, a gene complementing the mating and pseudohyphal growth defects of STE20 mutant yeast. View Abstract
Mos10 (Vps60) is required for normal filament maturation in Saccharomyces cerevisiae. View Abstract
Deficient natural killer cell cytotoxicity in patients with IKK-gamma/NEMO mutations. View Abstract
Detecting legionellosis by unselected culture of respiratory tract secretions and developing links to hospital water strains. View Abstract
Nonfilamentous C. albicans mutants are avirulent. View Abstract
Candida albicans strains heterozygous and homozygous for mutations in mitogen-activated protein kinase signaling components have defects in hyphal development. View Abstract
Locations